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Strong Effect Analgesic & Antipyretic 4-Acetamidophenol ( Paracetamol ) Powder

Categories Pharmaceutical Raw Materials
Brand Name: TINGYI
Model Number: CAS: 103-90-2
Certification: GMP, SGS , ISO 9001:2008 , KOSHER
Place of Origin: China
MOQ: 100g
Price: Negotiable ( Discounts For Big Order )
Payment Terms: Western Union, MoneyGram, Bank Transfer, Bitcoin
Supply Ability: 3000 KG/Month
Delivery Time: Within 7 Working Days
Packaging Details: Stealth And Discreet Packaging
Product Name: 4-Acetamidophenol
Other Name: Paracetamol
MW: 151.16
EINECS: 203-157-5
MP: 168-172 °C
FP: 11 °C
Density: 1.293 g/cm3
Water Solubility: 14 g/L (20°C)
Appearance: white crystalline solid
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    Strong Effect Analgesic & Antipyretic 4-Acetamidophenol ( Paracetamol ) Powder

    Strong Effect Analgesic & Antipyretic 4-Acetamidophenol (Paracetamol) Powder


    This product is acetanilide antipyretic analgesics. By inhibiting ring oxidase and

    selectivity in the hypothalamus of the temperature regulating center, the synthesis of

    prostaglandins in peripheral vascular expansion, sweating and antipyretic effect, strength of

    its antipyretic effect similar to aspirin.

    Paracetamol is a widely used over-the-counter analgesic (pain reliever) and antipyretic

    (fever reducer). Acetaminophen is the name adopted for this pharmacologic agent in the U. S.

    (USAN) and Japan; Paracetamol is approved in a variety of international venues.

    Paracetamol is classified as a mild analgesic. It is commonly used for the relief of

    headaches and other minor aches and pains and is a major ingredient in numerous cold and flu

    remedies. In combination with opioid analgesics, paracetamol can also be used in the

    management of more severe pain such as post-surgical pain and providing palliative care in

    advanced cancer patients. Though paracetamol is used to treat inflammatory pain, it is not

    generally classified as an NSAID because it exhibits only weak anti-inflammatory activity.


    Paracetamol is approved for reducing fever in people of all ages.

    Paracetamol is used for the relief of pains associated with many parts of the body. It has

    analgesic properties comparable to those of aspirin, while its antiinflammatory effects are

    weaker. It is better tolerated than aspirin in patients in whom excessive gastric acid

    secretion or prolongation of bleeding time may be a concern.

    Paracetamol powder nutritional supplemen Mainly used for amino acid infusion,due to special

    wettability, also used in cold cream,osmetics. Serine constitute the human body of several

    kinds of essential amino acid of protein a, for the construction of protein has a very

    important role.

    Research Result:

    Since their synthesis in the late 1800s paracetamol (acetaminophen) and phenacetin have

    followed divergent pathways with regard to their popularity as mild analgesic/antipyretic drugs.

    Initially, paracetamol was discarded in favour of phenacetin because the latter drug was

    supposedly less toxic. Today the opposite is true, and paracetamol, along with aspirin, has

    become one of the two most popular 'over-the-counter' non-narcotic analgesic agents. This

    marked increase in the wide approval attained by paracetamol has been accompanied by the

    virtual commercial demise of phenacetin because of its role, albeit somewhat circumstantial, in

    causing analgesic nephropathy. Both paracetamol and phenacetin are effective mild analgesics,

    suitable for treating mild to moderate pain, and their actions are broadly comparable with those

    of aspirin and related salicylates, although they do not appear to possess significant anti-

    inflammatory activity. Since a major portion of a dose of phenacetin is rapidly metabolised to

    paracetamol, it seems possible that phenacetin owes some of its therapeutic activity to its main

    metabolite, paracetamol, whereas its most troublesome side effect (methaemoglobinaemia) is

    due to another metabolite, p-phenetidine. The mechanism of action of paracetamol is poorly

    defined, although it has been speculated that it may selectively inhibit prostaglandin production in

    the central nervous system, which would account for its analgesic/antipyretic properties. The lack

    of any significant influence on peripheral cyclooxygenase would explain the absence of anti-

    inflammatory activity. At therapeutic doses paracetamol is well tolerated and produces fewer side

    effects than aspirin. The most frequently reported adverse effect associated with paracetamol is

    hepatotoxicity, which occurs after acute overdosage (usually doses greater than 10 to 15g are

    needed) and, very rarely, during long term treatment with doses at the higher levels of the

    therapeutic range. Paracetamol damages the liver through the formation of a highly reactive

    metabolite which is normally inactivated by conjugation with glutathione. Overdoses of

    paracetamol exhaust glutathione stores, thus allowing the accumulation of this toxic metabolite

    which covalently binds with vital cell elements and can result in liver necrosis. Glutathione

    precursors (notably intravenous N-acetylcysteine) have proved remarkably successful in treating

    paracetamol overdose, as long as treatment is initiated within 10 hours.

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